For clinicians · Medication guide

Allopurinol

allopurinol, Zyloprim

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Bottom lineAllopurinol is the strongly preferred first-line urate-lowering therapy for gout, including in moderate-to-severe CKD, started at a low dose and titrated to a serum urate target below 6 mg/dL.

Dosing and administration[2]

Oral, once daily after food for doses up to 300 mg; divide larger total daily doses (no single dose above 300 mg). Start low and titrate to a serum urate target with adequate hydration. Co-prescribe flare prophylaxis (colchicine or an NSAID) on initiation and for the first months. Consider HLA-B*5801 screening in higher-risk ancestries (Han Chinese, Thai, Korean) given the risk of severe cutaneous adverse reactions.

IndicationStarting doseTitration and maximum
Gout (urate lowering)Start 100 mg once daily (100 to 200 mg), increasing by about 100 mg at weekly intervals.Titrate against serum urate to below 360 micromol/L (6 mg/dL), or below 300 micromol/L (5 mg/dL) in tophaceous or severe gout. Usual maintenance 200 to 300 mg/day (mild) up to 400 to 600 mg/day (moderate to severe); maximum 800 mg/day. Start 50 to 100 mg/day or lower in renal impairment.

Onset. Serum urate normalises within about 1 to 3 weeks; adjust to target.

Renal adjustment. Reduce the dose in renal impairment (allopurinol and its active metabolite oxypurinol accumulate): with creatinine clearance about 10 to 20 mL/min use around 200 mg/day; below 10 mL/min do not exceed 100 mg/day; with severe impairment extend the dosing interval. Start low (50 to 100 mg/day or lower) and up-titrate slowly.

Hepatic. Use reduced doses and monitor liver function periodically in hepatic impairment.

Monitoring

Contraindications

Key interactions

Clinical notes

How it is used in gout

Xanthine oxidase inhibitor, strongly recommended as preferred first-line ULT for all patients including CKD stage 3 or worse, over all other agents. A xanthine oxidase inhibitor is also strongly preferred over probenecid in CKD stage 3 or worse.[1]

Before starting

HLA-B*5801 testing before starting allopurinol is conditionally recommended for patients of Southeast Asian descent (for example Han Chinese, Korean, Thai) and African American patients, who have a higher allele prevalence, and conditionally recommended against in others. The allele is strongly associated with allopurinol hypersensitivity syndrome.[1]

Monitoring

Serial serum urate guides dose titration to and maintenance of the target below 6 mg/dL (treat-to-target, strongly recommended). Specific on-treatment laboratory intervals are not set by the gout guideline; follow the product monograph and reconcile.[1]

Common effects

Background clinical knowledge (not from the guideline): rash, gastrointestinal upset, and transaminase elevation can occur; ULT initiation transiently increases flare risk, mitigated by anti-inflammatory prophylaxis.

Important cautions

Background clinical knowledge and monograph, not from the gout guideline: allopurinol hypersensitivity syndrome (including SJS/TEN and DRESS) is the key severe risk, higher early, with higher starting dose, and in renal impairment, which is why a low start is used. Allopurinol inhibits xanthine oxidase and markedly raises levels of azathioprine and 6-mercaptopurine, risking severe myelosuppression; avoid the combination or reduce the thiopurine substantially with close monitoring. Reconcile dosing and interactions with the product monograph.

References

  1. FitzGerald JD, Dalbeth N, Mikuls T, et al. 2020 American College of Rheumatology Guideline for the Management of Gout. Arthritis Care Res (Hoboken). 2020;72(6):744-760. doi:10.1002/acr.24180 https://doi.org/10.1002/acr.24180
  2. ALLOPURINOL (allopurinol tablets) Product Monograph. Health Canada authorized product monograph (Apotex), 2024. https://pdf.hres.ca/dpd_pm/00076177.PDF